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What Dihexa appears to do is bind to HGF itself and act as a co-agonist or superagonist at MET — potentiating HGF-mediated signaling rather than simply mimicking it
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Despite having overall lower efficacy, such dependence was not found for the velocity storage attenuation group (rho = −0.18) (Figure 3B, ①), and the group difference was statistically significant ( p = 0.024, Table 1)
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