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In relation to physiological significance, we conclude that the current notion that cf-mtDNA is pro-inflammatory is mostly derived from the misinterpretation of correlational clinical studies, as well as from experimental studies demonstrating how cytoplasmic (i.e., from inside the cell) mtDNA triggers antiviral signaling, and how exogenously applied purified forms of mtDNA potentiates but, in most cases, does not appear sufficient to systematically trigger inflammation

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