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Complementary Healing Pathways Beyond the receptor-ligand interaction, these peptides support healing through entirely different pathways that complement rather than overlap
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Prolonged expression of reactive microglia in KA-induced epilepsy contributes to the production of proinflammatory cytokines such as tumor necrosis factor alpha (TNF-α), interleukin-1β, and interleukin-6 resulting in neuronal loss and the promotion of epileptic seizures 24,25,26
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