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401 These findings suggest that Schwann cells under impaired metabolic conditions release pathologic EVs that promote nerve degeneration and DPN, whereas homeostatic EVs may facilitate nerve repair
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HIF-1α, as a transcriptional regulator plays a key role in the dysregulation of peroxisome proliferator-activated receptor alpha (PPARα) and Acyl-CoA, which ultimately hinders the production of FAO [37, 38]
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