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glutathione and male fertility improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, apoptosis induced by copper deposition in mice with Wilson disease cocomo-skin-c477150544118
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glutathione and male fertility improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, apoptosis induced by copper deposition in mice with Wilson disease cocomo-skin-c477150544118
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glutathione and male fertility improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, apoptosis induced by copper deposition in mice with Wilson disease cocomo-skin-c477150544118
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glutathione and male fertility improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, apoptosis induced by copper deposition in mice with Wilson disease cocomo-skin-c477150544118
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In the presence of GHK (Figure S16B–D), the amount of Aβ 1–16 still decreases over time, whereas the normalized areas related to the different carbonylated species increase as the reaction time increases (Figure S16A)
glutathione and male fertility improves testicular spermatogenesis through inhibiting oxidative stress, mitochondrial damage, apoptosis induced by copper deposition in mice with Wilson disease cocomo-skin-c477150544118
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